Traumatic Shoulder Instability.
Last updated Thursday, February 10, 2005
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Figure 5 - Functional impairment among TUBS patients
Figure 6 - Glenoid abutting against the cuff insertion to the head posteriorly
Evaluation and examination
Functional evaluation
The results from the Simple Shoulder Test
evaluations of patients with traumatic anterior glenohumeral
instability show that the most consistent functional impairment was the
inability to throw overhand, but many patients also had problems
sleeping, putting their hand behind their head, and lifting a gallon to
head level.Physical examination
The goal of the physical examination is largely to confirm the
impression obtained from the history: that a certain combination of arm
position and force application produces the actual or threatened
glenohumeral instability that is of functional concern to the patient.
If the diagnosis has been rigorously established from the history, for
example by documented recurrent anterior dislocations, it is not
necessary to risk redislocation on the physical examination. If such
rigorous documentation is not available, however, we must challenge the
ligamentous stability of the shoulder in the suspected position of
vulnerability. We seek to have the patient identify the positions and
events that are of functional significance.
The most common direction of recurrent traumatic instability is
anteroinferior. Stability in this position is challenged by externally
rotating and extending the arm elevated in the coronal plane. This is
conveniently done in the supine position with one of the examiner's
hands under the back of the shoulder serving as a fulcrum for the
external rotation and extension force. We apply extension and external
rotation loads in different positions of elevation to challenge the
various parts of the anterior capsular mechanism. The patient will very
often guard against the position of instability by actively limiting
the range of humeroscapular motion. For this reason, we may need to
hold the arm in the challenging position for 1 to 2 minutes to fatigue
the stabilizing musculature. When the muscle stabilizers tire, the
capsuloligamentous mechanism is all that is holding the humeral head in
the glenoid. At this moment the patient with traumatic anterior
instability becomes apprehensive, recognizing that the shoulder is
about to come out of joint. This recognition is strongly supportive of
the diagnosis of traumatic anterior instability.
Standard tests of glenohumeral laxity are of limited value in the
diagnosis of traumatic instability. As has been described earlier the
magnitude of translation on the standard test of glenohumeral laxity
does not distinguish stable from unstable shoulders. The magnitude of
translation on some clinically stable shoulders may be as great or
greater than that on shoulders with traumatic instability. However, the
experienced examiner may detect certain findings such as increased ease
of translation on the anterior drawer test or grinding as the humeral
head slides over a bony edge of the glenoid from which the labrum has
been avulsed. There may also be a catching or a locking of a torn
glenoid labrum, producing findings analogous to those of a torn
meniscus in the knee.
We do not consider pain on abduction, external rotation and
extension to be specific for instability. Such pain may relate to
shoulder stiffness or alternatively to abutment of the glenoid against
the cuff insertion to the head posteriorly. Furthermore, relief of this
pain by anterior pressure on the humeral head may result from
diminished stretch on the anterior capsule or from relief of the
abutment posteriorly.
In all patients with traumatic instability but particularly in those
over age 35, the strength of the internal and external rotation must be
examined. With increasing age, there is an increasingly common
association between traumatic glenohumeral instability and rotator cuff
defects. Patients with significant cuff lesions may demonstrate atrophy
of the spinatus muscles as well as pain and or weakness on resisted
abduction or external rotation. Any rotator cuff pathology must be
recognized and incorporated in the treatment plan.
Finally, carefully examine the brachial plexus to assure that the
episodes of traumatic instability have not compromised its function.
Radiographs
Radiographs frequently help to provide confirmation of traumatic
glenohumeral instability. These findings may include an indentation or
impaction in the posterior aspect of the humeral head from contact with
the anteroinferior corner of the glenoid when the joint is dislocated.
Radiographs may also reveal a periosteal reaction to the ligamentous
avulsion at the glenoid lip or occasionally a fracture of the glenoid
rim.
These lesions are usually revealed by an anteroposterior view in the
plane of the scapula, a proper axillary view, and an apical oblique
view, as shown in the example movie listed below. We have not found
additional radiographic views, CT arthrography, or MRI to be cost
effective in the evaluation of shoulders with characteristic traumatic
instability. We occasionally use CT to define the magnitude of bone
loss when a sizable humeral head or glenoid defects have been
identified on a series of plain radiographs.
In a patient whose onset of traumatic instability occurred after age
35 there may be evidence on history and physical examination of rotator
cuff pathology. In these situations, preoperative imaging of cuff
integrity may play an important role in surgical planning: the approach
for rotator cuff repair is quite different than the approach for the
repair of an anterior inferior capsular lesion.
Electromyography may be helpful in the evaluation of the patient
with recurrent traumatic instability if the history and physical
examination suggest residual brachial plexus lesions.
Movie
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