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HomeAbout traumatic instabilitySummaryAssociated pathologiesTUBS syndromeSLAP lesionsHistoryEvaluation and examinationTreatment

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Traumatic Shoulder Instability.

Last updated Thursday, February 10, 2005

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Figure 1 - Torque equilibrium
Figure 1 - Torque equilibrium

Figure 2 - Anterior glenohumeral ligament
Figure 2 - Anterior glenohumeral ligament

Figure 3 - Glenoid abutts against the insertion of the cuff
Figure 3 - Glenoid abutts against the insertion of the cuff

About traumatic instability

What is traumatic glenohumeral instability (TUBS)?

Traumatic instability (TUBS) is instability that arises from an force large enough to injure some of the major supporting structures of the joint, such as the glenohumeral capsule, ligaments, rotator cuff, or the bone of the humerus or glenoid.

Usually, traumatic instability arises from a fall on the outstretched hand, for example in skiing, football, or wrestling. In addition to a careful history and clinical examination, X-rays may be helpful in evaluating the patient with traumatic instability.

When the instability happens repeatedly or when fear of recurrent dislocation interferes with normal use of the shoulder, surgical repair of the injury can be considered. The postoperative care after this surgery is very important.

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What are the associated pathologies of traumatic instability of the shoulder?

The most common pathology associated with traumatic instability is the avulsion of the anteroinferior capsule and ligaments from the glenoid rim. Substantial force is required to produce this avulsion in a healthy shoulder. While this load may be applied directly (for example, by having the proximal humerus hit from behind), an indirect loading mechanism is more common. Indirect loading is most easily understood in terms of a simple model of the torques involved. When the upper extremity is abducted and externally rotated by a force applied to the hand, the following equation for torque equilibrium is a useful approximation, if we attribute the major stabilizing role to the ligament: I x R = F x A or equivalently I = F x A/R, where "I" is the tension in the inferior glenohumeral ligament, "R" is the radius of the humeral head, "F" is the abduction external rotation load applied to the hand, and "A" is the distance from the center of the humeral head to the hand. If the radius of the humeral head is 2.5 cm and the distance from the head center to the hand is one meter, this formula suggests that the inferior glenohumeral ligament would experience a load 40 times greater than that applied to the hand. From this example we can see that a much lesser load is required to produce the characteristic lesion of traumatic instability if this load is applied indirectly through the lever arm of the upper extremity.

Avulsion of the anterior glenohumeral ligament mechanism deprives the joint of stability in positions where this structure is a major stabilizer of the joint, which is typically approaching maximal external rotation and extension of the arm elevated near the coronal plane. These are positions in which stability is dependent on integrity of the capsuloligamentous mechanism. Thus, it is evident that in recurrent traumatic instability, problems are most likely to occur when the arm is placed in a position approximating that in which the original injury occurred. Midrange instability may also result from a traumatic injury if the glenoid concavity is compromised by avulsion of the labrum or fracture of the bony lip of the glenoid. Lessening of the effective glenoid depth compromises the effectiveness of concavity compression, reduces the angles through which the glenoid can balance the net joint reaction force, reduces the surface available for adhesion-cohesion, and compromises the ability of the glenoid suction cup to conform to the head of the humerus.

The corner of the glenoid abuts against the insertion of the cuff to the tuberosity when the humerus is extended, abducted, and externally rotated. Thus, the same forces involved in challenging the strength of the inferior glenohumeral ligament are also applied to the greater tuberosity-cuff insertion area. It is not surprising, therefore, that tuberosity fractures and cuff injuries may be a part of the clinical picture of traumatic instability. The exact location and type of traumatic injury depends on the age of the patient and the magnitude, rate, and direction of force applied. Avulsions of the glenoid labrum and glenoid rim fractures are more commonly seen in young individuals after a major injury. In patients over the age of 35, traumatic instability tends to be associated with fractures of the greater tuberosity and rotator cuff tears. This tendency increases with increasing age at the time of the initial traumatic dislocation. Thus, as a rule, younger patients require management of anterior lesions and older patients require management of posterior lesions.

The posterior lateral humeral head defect is a common feature of traumatic instability. These lesions are often noted after the first traumatic dislocation and tend to increase in size with recurrent episodes. This impaction injury occurs when the anterior corner of the glenoid is driven into the posterior lateral humeral articular surface. It is evident that this injury is close to the cuff insertion. Large head defects compromise stability by diminishing the articular congruity of the humerus.

What does TUBS mean?

We refer to the usual type of traumatic instability as the TUBS syndrome because it arises from a significant episode of Trauma, characteristically from abduction and extension of the arm elevated in the coronal plane. The resulting instability is usually Unidirectional in the anteroinferior direction. The pathology is usually an avulsion of the capsuloligamentous complex from the anterior inferior lip of the glenoid, commonly referred to as a Bankart lesion. With functionally significant recurrent traumatic instability, a Surgical repair of this ligament avulsion is frequently required to restore function. We have established some diagnostic criteria for making this diagnosis.

What are SLAP (superior labrum, anterior to posterior) lesions and how are they managed?

SLAP lesions (superior labrum, anterior to posterior) are detachments of labrum (the lining of the glenoid socket of the shoulder). In many cases it is difficult to tell if the labral attachment is normal or not--even with arthroscopy. Thus it is important that the surgeon and the patient be able to relate the symptoms to the finding on arthroscopy. This is a particular challenge because there are no symptoms or presurgical tests that reliably establish the presence of a symptomatic SLAP lesion.

Surgery can either trim the labrum or attempt to reattach it.

After this surgery, the patient experiences the same type of discomfort as after other arthroscopic surgeries. The details of the recovery and rehabilitation depend on the specifics of the surgery. For example, if there is a trimming, early motion may be carried out, while, after a repair, a period of restricted use may be in order until healing has occurred.


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