Shoulder and Elbow Cases to Consider.
Last updated Friday, February 11, 2005
Figure 1 - Destruction of the humeral head with an associated soft tissue mass Figure 2 - Cervicothoracic syrinx Unconstrained total elbow arthroplasty for rheumatClinical presentation This is the radiograph of a 60 year old woman with chronic
rheumatoid arthritis who presented to us with problems 4 weeks after
left total elbow arthroplasty. She had a similar procedure performed on
the opposite side without problem. One week after this surgery she felt
a "clunk" in her elbow and then had difficulty moving it without
discomfort. Radiographs showed the elbow had dislocated (see figure 1).
A closed reduction was performed, but 2 weeks later the dislocation
recurred. Open reduction and a soft tissue reconstruction was performed
at that time. Now the elbow is again dislocated, swollen and painful on
all motion--she keeps it in a splint at all times. Her lateral elbow
incision is relatively calm, but the sutures are still in place. She
has had two surgeries and one manipulation in less than one month. Her
exam indicates ulnar nerve irritation, but she is otherwise
neurovascularly intact.
Our concerns included:
- the patient's loss of elbow function
- wound status
- neurovascular status
- risks of revision of cemented prosthesis in soft rheumatoid bone
- incisional approaches
Management Closed reduction was attempted under anesthesia and fluoroscopy. This could not be accomplished.
Open reduction was attempted after ulnar nerve dissection through a
new posteriormedial approach. A stable reduction could not be achieved.
Revision to constrained total elbow was accomplished with minor
penetrations of ulna and humerus in process of cement removal. Post
operative range was 0-135 degrees. Neurovascular status intact. See
post operative radiograph (figure 2). Clinical presentation This is the radiograph of the right shoulder of a 50 year old woman
who presented with a chronic atrophic non-union of her humerus (see
figure 1). She sustained the subtuberous fracture in a fall 6 months
ago. She was treated with closed reduction and sling immobilization. At
6 weeks mobilization was started, however over the ensuing months she
had progressively increasing pain in her arm.
At her consultation visit with us, examination revealed pain and
crepitance on movement of the arm. There was no evidence of sepsis or
neurovascular impairment. Her general health was excellent.
Our concerns included:
- The loss of bone around the fracture site.
- The local osteopenia.
- The method of internal fixation (if a prosthesis was not used).
- The challenge of obtaining union between the tuberosities and humeral shaft if a prosthesis was used.
Management In our view the primary problem here was not the articular surface
nor the length of the bone, but rather the challenge of getting the
tuberosities to heal to the shaft. We elected a method of treatment
which respected the compromised bone quality and which maximized the
contact between bone of the proximal and distal fragments. On these
bases, neither interpositional bone graft, metallic internal fixation
nor a prosthesis was used. The shoulder was approached through a
deltopectoral incision to protect the deltoid muscle. The distal aspect
of the proximal fragment was carefully carved to receive and interlock
with the proximal end of the distal fragment. The insertion of the peg
of the humeral shaft in to the hole in the head was secured with six
large (#5) non nonabsorbable sutures passed through holes in the
proximal shaft and then through the proximal humeral metaphysis, out
the cuff insertion and around the tuberosities. Iliac crest autograft
was added around the non-union site. The fixation was robust, so early
gentle active motion was started immediately.
Radiographs taken 6 months later show a united fracture (see figure
2). The arm is one inch short and the deltoid lag is resolved. The
patient is now pain free, has good use of the shoulder, and is pleased. Clinical presentation This is the radiograph of a 25 year old male with a history of
recurrent anterior dislocations of his right shoulder (see figure 1).
His original dislocation occurred 5 years ago following a seizure.
Unfortunately over the ensuing two years his seizures were poorly
controlled resulting in further dislocations. Over the last three years
he has had two surgical procedures (a Bankart repair and a revision
Bankart repair with soft tissue augmentation), but continues to have
instability whenever his arm is brought into abduction and minimal
external rotation. Due to the dislocations he is unable to work or
perform normal daily activities above shoulder level. His epilepsy is
well controlled on medication.
On examination the patient was very apprehensive with his arm in
abduction and in thirty degrees of external rotation. With his arm by
the side he could externally rotate to sixty degrees without discomfort
in comparison to eighty degrees on the other side.He had a functioning
rotator cuff and subscapularis. He had no evidence of ligamentous
laxity,a negative sulcus sign and a negative jerk test.
Our concerns include:
- Significant deficiency of the anterior / inferior glenoid.
- Early degenerative change of the glenohumeral joint.
- The history of seizures.
- The failure of two previous repairs.
Management Despite early degenerative changes on X-ray, the patient's primary
functional problem was instability. Examination under anesthesia
revealed that there was no effective anterior glenoid lip on the load
and shift test. Surgical findings confirmed the lack of an anterior
inferior glenoid lip, a large recurrent Bankart lesion, a large
Hill-Sachs defect, and early degenerative changes. The subscapularis
was intact.
We concluded that a robust reconstruction of the anterior glenoid
was essential to stabilizing this shoulder and that the patient's
history demonstrated that this could not be accomplished with soft
tissue procedures. Thus we reconstructed the anterior glenoid using a
contoured iliac crest graft with capsule interposed between the graft
and the humeral head.
One year following surgery, the bone graft remains stable (see
figure 2) and the patient uses the shoulder for daily activities
without apprehension, instability, or complaints of pain. Clinical presentation This is the radiograph of a fifty year old woman with rheumatoid
arthritis who presented to our unit with a complex shoulder history
(see figure 1). Six years previously she underwent a right total
shoulder replacement and did well until four years later, when she
fractured her humerus below the stem of the prosthesis in an automobile
accident. The fracture was treated with open reduction and internal
fixation of the humerus using a plate and screws. Postoperatively,
however, the arm became infected with staph aureus, requiring removal
of the internal fixation and prosthesis and the insertion of
antibiotic-impregnated cement beads around and within the humeral
shaft. At the time of presentation to us, the patient's chief complaint
was pain and inability to use her arm due to weakness and instability.
She answered "no" to all 12 questions of the Simple Shoulder Test.
There was no clinical evidence of infection, a sedimentation rate of
25, and the shaft fracture appeared healed. Her main question was
whether a new joint could be re-inserted.
Our concerns include:
- Had the humeral shaft fracture united?
- Was there still residual infection?
- Was it possible to revise this to a hemiarthroplasty?
- Could a humeral stem be inserted all the way down the humeral shaft?
Management After thorough discussion of the risks with the patient, she decided
to have us explore the shoulder with the possibility of inserting a
prosthesis if there was no evidence of sepsis. Without preoperative
antibiotics, the shoulder was exposed through the previous
deltopectoral incision. A large amount of residual scar tissue was
identified yet there was no macro or microscopic evidence of underlying
sepsis. After cultures were obtained, intravenous antibiotics were
administered. The shaft appeared to be solidly healed. Although our
initial plan was to insert a long stemmed component; removal of the
beads from the intact shaft proved impossible. Rather than splitting
the humerus to remove the remaining beads, we cut down the stem of a
prosthesis to fit the available space in the medullary canal. A solid
press fit was achieved without additional cement. This prosthesis
provided a smooth and stable articulation with the remaining glenoid.
Forty-eight hours after surgery the patient was discharged with a
comfortable shoulder which she could elevate to 140 degrees and
externally rotate to forty degrees. She was pleased and already more
functional than at the time of admission. Clinical presentation This is the radiograph of a 70 year old woman with
spondylo-epiphyseal dysplasia who has had severe limitation of her
right shoulder motion since birth. Over the last few years she has
progressively experienced severe pain which prevents her from using
this arm and from sleeping. She also uses a wheel chair for ambulation
because of dysplastic involvement of her spine, hips and knees. Her
left shoulder remains relatively unaffected.
On examination her right shoulder is relatively fixed in internal
rotation. Any use or motion of the joint is painful for her and
produces crepitance. Her attitude is very positive.
Our concerns include :
- Marked disuse osteopenia.
- The technical difficulty of a shoulder arthroplasty.
- The potential for improved function after an arthroplasty due to the chronicity of her condition.
- The feasibility and potential benefit of an arthrodesis.
Management The patient clearly identified pain at rest and on use of her
shoulder as her primary problem; she had thoroughly adapted to the loss
of motion. Because of her serviceable left arm and her need for a
strong right shoulder for transfers and wheel chair ambulation, we
recommended a shoulder arthrodesis using internal fixation for
immediate return to function.
At surgery the virtual absence of glenohumeral motion was verified.
The shoulder was fused in the same position it had assumed for 70
years: slight flexion and abduction with 45 degrees of internal
rotation. The arthrodesis was secured with a pelvic reconstruction
plate. She began using her arm on the first post operative day. Three
months after surgery the shoulder was essentially painless and allowed
function in the necessary activities of daily living. Clinical presentation This is the radiograph of a 45 year old man who presented to our
clinic with a painful left shoulder of 5 months duration (see figure
1). His chronic anterior dislocation apparently resulted from a fall
after an epileptic seizure. An unsuccessful attempt had been made 1
month earlier to reduce the dislocation in the emergency room. His
current complaint was disabling pain and limited range of motion.
On examination the patient held his right shoulder in fixed internal
rotation. Obvious asymmetry was present around the shoulder girdle when
compared to the contralateral side. His humeral head could be palpated
anteriorly and inferiorly and any form of shoulder movement caused
severe pain. His axillary nerve was functioning and his remaining
neurovascular examination was normal. The patient had a history of
alcoholism and epilepsy poorly controlled by medication.
Our concerns include :
- The patients' general health and well-being.
- The possibility of maintaining a reduction following an open procedure.
- The condition and viability of his humeral head.
- If a hemiarthroplasty was necessary, what soft tissues would maintain stability in this patient.
- Would there be a need for a bony procedure around the glenoid to establish stability?
Management Despite the patient's history of alcoholism and poorly controlled
epilepsy we thought it appropriate to try to treat the patient and his
shoulder due to his chronic disabling pain.
No attempt was made to perform a closed reduction due to the chronicity of the dislocation.
A routine deltopectoral incision was made exposing the subscapularis
and dislocated humeral head. Care was then taken to identify and
protect the axillary nerve. It was impossible to openly reduce the
humeral head due to a large Hill Sachs lesion involving at least 50% of
the articular surface and severe contracture of the posterior
structures.
Due to the humeral head destruction yet an intact glenoid, a
decision was made to perform a hemiarthroplasty rather than a TSA. An
extensive release of the posterior capsule and cuff was performed
before inserting the humeral component. This was then reduced with
difficulty and showed signs of anterior subluxation when the arm was
rotated away from neutral. The component was therefore placed in some
45 degrees of retroversion and a more extensive posterior release was
performed around the glenoid. We did not feel the necessity to place an
anterior bone block on the glenoid although this would be a possibility
if instability persisted.
Due to concerns of patient compliance and the potential for
instability post-operatively he was placed in a shoulder immobilizer
for 6 weeks.
The current radiographs are shown demonstrating a located humeral
head and a small amount of heterotopic ossification which commonly
occurs following a chronic dislocation (see figure 2). No further
episodes of instability have occurred at 6 months follow-up and the
patient is currently satisfied with the result. Clinical presentation This is the radiograph of a 50 year old, otherwise normal woman who
presented with a comminuted fracture of her proximal humerus sustained
in a fall (see figure 1). She had a normal neurovascular examination.
Our concerns include :
- Could this fracture be successfully managed non-operatively?
- If operative intervention were necessary, what would be the best method of internal fixation?
Management The fracture was managed nonoperatively. At 6 weeks the radiographs
(see figure 2) showed early callous and the position remained
acceptable so gentle motion was commenced. Twelve months later the
fracture was solidly united. She had full use of her arm and a full
range of movement both of her shoulder and elbow.
Clinical presentation This 45 year old female presented with a one-year history of
gradually increasing left shoulder pain and gradual loss of range of
motion (see figure 1). The patient recalled an injury about one year
prior to presentation when some boxes she was carrying were forced into
her shoulder by a closing door.
On examination no soft tissue mass was present around her shoulder
region.There was tenderness to palpation over her anterior deltoid.
There was loss of motion of the affected shoulder particularly
involving external rotation and forward elevation. Her neurovascular
examination was unremarkable.
Her general health and overall physical examination was normal expect for a past history of benign thyroid nodules.
Our concerns include :
- Establishing an accurate diagnosis of the bony / soft tissue lesion.
- What further investigations were necessary?
- Possible management strategies depending on the diagnosis.
Management Our differential diagnosis from history and radiographic appearance
included myositis ossificans, high grade surface osteosarcoma, and
parosteal osteosarcoma. Further studies used to help differentiate and
stage the lesion included a CT scan of her shoulder and chest, MRI of
her shoulder, technetium bone scan and appropriate blood workup.
Following these studies an open biopsy was performed through the
anterior edge of the deltoid which could be later sacrificed if a
formal resection was required. Biopsy confirmed the diagnosis of
"parosteal osteosarcoma".
The options of management were then discussed at length with the patient and included:
- Limb salvage via a proximal humeral allograft
- Shoulder disarticulation
- Tikhof-Lindberg
- Allograft arthrodesis
- Mega-prosthesis
A mutual decision was made to perform a limb salvage procedure via a
proximal humeral allograft and a cemented long stem Neer prosthesis as
seen in the current radiograph (see figure 2). This was performed
through an extended anterior deltopectoral approach. Adequate margins
were achieved by resecting the proximal 1/3 of the humerus including
part of the cuff and part of deltoid, pectoralis major, latissimus
dorsi, and teres major. Neurovascular structures were able to be
preserved.
The remaing cuff and capsule were reattached to the allograft. The
deltoid insertion to the humerus was preserved. Microscopic pathology
revealed adequate margins. Clinical presentation A 43 year old right-hand-dominant male, with a history of a
nonspecific injury to his left shoulder while skiing 15 years ago,
presented to the University of Washington Shoulder and Elbow Service
complaining of crepitus in his left shoulder. Approximately 3 to 4
weeks prior to presentation, the patient noted acute onset of painless
and progressive left shoulder swelling. The patient's past medical and
surgical histories were otherwise unremarkable.
Physical examination was remarkable for a 10 x 15 cm nontender mass
in the anterolateral aspect of his left shoulder. Neurovascular
examination was remarkable only for a vague, nondermatomal decrease in
pinprick sensation in his left upper extremity. Proprioception and
vibration sensation were preserved.
Plain radiographs of the left shoulder revealed destruction of the
humeral head with an associated soft tissue mass (see figure 1).
Laboratory tests were unremarkable. Management The differential diagnosis included Charcot joint, septic arthritis,
and neoplasm. The patient was taken to the operating room for open
biopsy of his left shoulder mass. Cultures were negative. Histological
examination showed chronic inflammation, reactive new bone formation,
and fibrosis suggestive of neuropathic arthropathy. Subsequently, the
patient had an MRI of the cervical spine which demonstrated a
cervicothoracic syrinx (see figure 2).
The patient was evaluated by the Neurosurgery Service and underwent a syringoperitoneal shunt.
Since arthroplasty and arthrodesis in this group of patients have a
historically high failure rate, no further surgical intervention in
planned. The patient has been educated as to the disease process as
well as conservative treatment to maximize his function.
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