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HomeAbout recurrent shoulder instabilityRecurrent atraumatic instabilityThe history (atraumatic)The physical examination (atraumatic)Recurrent traumatic instabilityTUBSThe history (traumatic)The physical examination (traumatic)

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Evaluation of Recurrent Instability.

Last updated Friday, November 16, 2007

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Figure 6
Figure 6

Figure 7
Figure 7

Figure 8
Figure 8

Figure 9
Figure 9

Recurrent traumatic instability

Traumatic instability is instability that arises from an injury of sufficient magnitude to tear the glenohumeral capsule, ligaments, labrum or rotator cuff or to produce a fracture of the humerus or glenoid.

TUBS

A typical patient is a 17-year-old skier whose recurrent anterior instability began with a fall on an abducted, externally rotated arm (although the condition has been reported in individuals as young as three years old. (Endo et al, 1993) In order to injure these strong structures, a substantial force must be applied to them. The most common pathology associated with traumatic instability is the avulsion of the anteroinferior capsule and ligaments from the glenoid rim. Substantial force is required to produce this avulsion in a healthy shoulder. While this load may be applied directly (for example, by having the proximal humerus hit from behind), an indirect loading mechanism is more common. Indirect loading is most easily understood in terms of a simple model of the torques involved. When the upper extremity is abducted and externally rotated by a force applied to the hand, the following equation for torque equilibrium is a useful approximation, if we attribute the major stabilizing role to the ligament (see figure 6): T = B * E/R where "T" is the tension in the inferior glenohumeral ligament, "R" is the radius of the humeral head, "B" is the abduction external rotation load applied to the hand, and "E" is the distance from the center of the humeral head to the hand. If the radius of the humeral head is 2.5 cm and the distance from the head center to the hand is one meter, this formula suggests that the inferior glenohumeral ligament would experience a load 40 times greater than that applied to the hand. From this example we can see that a relatively small load is required to produce the characteristic lesion of traumatic instability if this load is applied indirectly through the lever arm of the upper extremity.

Avulsion of the anterior glenohumeral ligament mechanism (see figure 7) deprives the joint of stability in positions where this structure is a check rein, such a in maximal external rotation and extension of the arm elevated near the coronal plane. Thus, it is evident that in recurrent traumatic instability, problems are most likely to occur when the arm is placed in a position approximating that in which the original injury occurred. Midrange instability may also result from a traumatic injury because the glenoid concavity may be compromised by avulsion of the labrum or fracture of the bony lip of the glenoid (see figure 8). Lessening of the effective glenoid arc compromises the effectiveness of concavity compression, reduces the balance stability angles, reduces thesurface available for adhesion-cohesion, and compromises the ability of the glenoid suction cup to conform to the head of the humerus.

The corner of the glenoid abuts against the insertion of the cuff to the tuberosity when the humerus is extended, abducted, and externally rotated (see figure 9). (Liu and Boynton, 1993; Matsen, Lippitt, Sidles et al, 1994; Montgomery and Jobe, 1994; Rossi et al, 1994; Walch et al, 1991; Walch et al, 1993) Thus, the same forces which challenge the inferior glenohumeral ligament are also applied to the greater tuberosity-cuff insertion area. It is not surprising, therefore, that posterolateral humeral head defects, tuberosity fractures and cuff injuries may be a part of the clinical picture of traumatic instability. The exact location and type of traumatic injury depends on the age of the patient and the magnitude, rate, and direction of force applied. Avulsions of the glenoid labrum, glenoid rim fractures and posterolateral humeral head defects are more commonly seen in young individuals. In patients over the age of 35, traumatic instability tends to be associated with fractures of the greater tuberosity and rotator cuff tears. This tendency increases with increasing age at the time of the initial traumatic dislocation. Thus, as a rule, younger patients require management of anterior lesions and older patients require management of posterior lesions.

The posterior lateral humeral head defect is a common feature of traumatic instability. These lesions are often noted after the first traumatic dislocation and tend to increase in size with recurrent episodes. This impaction injury usually occurs when the anterior corner of the glenoid is driven into the posterior lateral humeral articular surface. It is evident that this injury is close to the cuff insertion. Large head defects compromise stability by diminishing the articular congruity of the humerus.

To help recall the common aspects of traumatic instability, we use the acronym TUBS. The instability arises from a significant episode of Trauma, characteristically from abduction and extension of the arm elevated in the coronal plane. The resulting instability is usually Unidirectional in the anteroinferior direction. The pathology is usually an avulsion of the labrum and capsuloligamentous complex from the anterior inferior lip of the glenoid, commonly referred to as a Bankart lesion. With functionally significant recurrent traumatic instability, a Surgical reconstruction of this labral and ligament avulsion is frequently required to restore stability.

The reader is referred to an review of the pathology and pathogenesis of traumatic instability by Wirth and Rockwood. (Wirth and Rockwood, 1993)


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