Orthopaedics & Sports Medicine  
  Home   |   Site Map   |   Contact Us   |   Links   |   News  
Orthopaedics & Sports Medicine  
Advanced Search
Orthopaedics & Sports Medicine
HomeAboutIncidence and risk factorsSymptomsAnatomy Symptoms Causes and effects Management and treatmentResearchConclusion

Print Print Complete Article
View article with questions Hide Questions



Osteoarthritis.

Last updated Thursday, February 10, 2005

<< Previous Page Next Page >>

Figure 3 - Bouchard's nodes
Figure 3 - Bouchard's nodes

Figure 4 - Heberden's nodes
Figure 4 - Heberden's nodes

Figure 1 - Healthy joint
Figure 1 - Healthy joint

Figure 2 - Joint with severe osteoarthritis
Figure 2 - Joint with severe osteoarthritis

Symptoms

What parts of the body does osteoarthritis affect?

In the hip, DJD may produce pain around the groin or in the inner thigh. Some people feel referred pain to the buttocks, the knee or along the side of the thigh. Degenerative joint disease of the hip may cause a limp and may limit range of motion, for example making it difficult to spread the legs.

Degenerative joint disease of the knees may produce pain and stiffness of the knee associated with a grating or catching sensation in the joint when it is moved. It may make it difficult to walk up and down stairs and lumps may be noted particularly along the medial (inner side) of the knee. If the pain prevents you from moving or exercising your knee, the large muscles around the knee area will become weaker.

Degenerative joint disease of the fingers may produce bony lumpiness around the joints of the finger and perhaps pain and stiffness of these joints as well. In the fingers: The breakdown of joint tissue in the fingers causes bony growths (spurs) to form in these joints. If spurs occur in the end joints of the fingers, they are called Heberden's nodes. If they occur in the joints in the middle of the fingers they are called Bouchard's nodes (see figures 3 and 4).

Degenerative joint disease of the feet most commonly affects the large joint at the base of the big toe. Stiffness, lumpiness and pain may be associated. Wearing tight shoes and high heels can make this pain worse.

Degenerative joint disease of the spine may produce stiffness of the back and at times, symptoms of pressure on the spinal cord and nerves running through the spine. The latter are particular important to notice and may include numbness or weakness of the arms or legs, difficulty with controlling bowel or bladder, loss of balance and pain radiating out the arms or down the legs.

Bone spurs

Bone spurs are of two basic types. One is the kind that arises near a joint with osteoarthritis or degenerative joint disease. In this situation, the cartilage has been worn through and the bone responds by growing extra bone at the margins of the joint surface. These "spurs" carry the formal name "osteophytes." They are common features of the osteoarthritic shoulder, elbow, hip, knee and ankle. Removing these osteophytes is an important part of joint replacement surgery, but removing them without addressing the underlying arthritis is usually not effective in relieving symptoms.

The second type of bone spur is the kind that occurs when the attachment of ligaments or tendons to bone become calcified. Thus can occur on the bottom of the foot, around the Achilles Tendon, and in the coroacoacromial ligament of the shoulder. Thus spurs often look impressive on X-rays, but because they are in the substance of the ligaments, rarely cause sufficient problems to merit excision.

What are symptoms of osteoarthritis?

Osteoarthritis is characterized by clinical symptoms of joint pain and aching, limited range of motion and instability, radiographic evidence of the erosion of the articular cartilage, joint space narrowing, sclerosis of the subchondral bone, and osteophytes (spurs). Other symptoms include stiffness and roughness on motion; these symptoms are worse after heavy use.

OA pathological changes involve both the cartilage and the bones. Until about 20 years ago, OA was widely assumed to reflect the passive erosion of the bearing surfaces of cartilage in the joints. Degeneration of the articular cartilage was viewed as a normal aging process much like old tires on a car wearing themselves out after extensive usage. This view is rapidly changing. Degeneration and loss of the articular cartilage in osteoarthritis is explained in terms of the pathophysiologic processes involved in the metabolism of cartilage rather than the inevitable mechanical wear and tear due to aging.

If degenerative joint disease is related to abnormalities of articular cartilage, it may involve many of the joints of the body. On the other hand, if the degenerative joint disease is caused by an injury, only one joint may be involved. The hips, knees, spine, and shoulders are most commonly involved. This condition may also affect some finger joints, the joint at the base of the thumb, and the joint at the base of the big toe.

The typical joint changes in osteoarthritis can be seen by comparing a healthy joint with a joint with osteoarthritis (see figures 1 and 2). In osteoarthritis, the normally smooth cartilage surface softens and becomes pitted and frayed. As the cartilage breaks down, the joint may lose its normal shape. The bone ends thicken and form bony growths, or spurs, where the ligaments and capsule attach to the bone.

Stiffness and joint deformity usually progress slowly without general body symptoms. By contrast, rheumatoid arthritis (RA) usually begins earlier, often developing more suddenly. RA usually affects same joint on both sides of body (e.g. both knees), causing redness, warmth, and swelling of many joints. RA is often accompanied by a general feeling of sickness, fatigue, weight loss, and fever.


<< Previous Page Next Page >>


How useful was this page or article?

This article is rated ***0.74 out of 5 stars (533 ratings).

Not useful at all Not very useful Useful Very useful Extremely useful
* ** *** **** *****
Team Physicians to the UW Huskies Varsity Athletes...And You!
Copyrights and disclaimer  | Privacy statement | Editorial policy
Problems or questions? Contact the webmaster.
Copyright © 2008 University of Washington - Seattle, WA. All rights reserved.