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HomeHistoryPhysical examinationRadiographic evaluationDisease characteristicsDegenerative joint diseasePrimary DJDSecondary DJDRheumatoid and other types of inflammatory arthritCuff tear arthropathyCapsulorrhaphy arthropathyAvascular necrosisOther types of arthritis

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Clinical Presentation and Evaluation of Glenohumeral Arthritis.

Last updated Thursday, January 27, 2005

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Figure 4
Figure 4

Degenerative joint disease

Primary DJD

In degenerative joint disease (DJD), the glenoid cartilage and subchondral bone are typically worn posteriorly, often leaving intact articular cartilage anteriorly (see figure 4). The cartilage of the humeral head is eroded in a "Friar Tuck" pattern of central baldness, often surrounded by a rim of remaining cartilage and osteophytes. Degenerative cysts may occur in the humeral head or glenoid. Osteophytes typically surround the anterior, inferior and posterior aspects of the humeral head and the inferior and posterior glenoid. As a result the humeral and glenoid articular surfaces have a flattened configuration which blocks rotation. Loose bodies are often found in the axillary or subscapularis recesses. The triad of anterior capsular contracture, posterior glenoid wear and posterior humeral subluxation is common in primary degenerative joint disease. Rotator cuff defects are uncommon in primary degenerative joint disease.

Secondary DJD

In contrast to primary degenerative joint disease, secondary degenerative joint disease arises when previous injury, surgery or other condition affects the joint surface precipitating its degeneration. In chronic, unreduced dislocations, (Hawkins, Neer, Pianta, et al., 1987; Pritchett and Clark, 1987; Rowe and Zarins, 1982) the humeral head may be indented and worn. The cartilage of the joint surfaces may be replaced with scar, or the subchondral bone may be so weakened by bone atrophy that it will collapse after reduction, leading to an incongruous joint surface. Samilson (Samilson and Prieto, 1983) identified seventy-four shoulders with a history of single or multiple dislocations that exhibited radiographic evidence of glenohumeral arthritis. The dislocations had been anterior in sixty-two shoulders, posterior in eleven, and one had multidirectional instability. The number of dislocations was not related to the severity of the arthrosis. Shoulders with posterior instability had a higher incidence of moderate or severe arthritis, as did shoulders with previous surgery in which internal fixation devices intruded on the joint surface. Hawkins and his co-workers (Hawkins, Neer, Pianta, et al., 1987) have suggested hemiarthroplasty if the dislocation is greater than six months old or if the humeral head defect involves more than 45% of the articular surface. If the glenoid is destroyed, a total shoulder arthroplasty may be indicated.

Tanner and Cofield reviewed twenty-eight shoulders with chronic fracture problems requiring prosthetic arthroplasty. (Tanner and Cofield, 1983) Sixteen had malunions with a joint incongruity, eight had post-traumatic osteonecrosis, and four had nonunion of a surgical neck fracture with a small, osteopenic head fragment.

Shoulders with secondary degenerative joint disease often present complex pathology and difficult surgical management. (Huten and Duparc, 1986; Neer and Kirby, 1982) Difficulties may be related to a number of factors: muscle contracture, scarring, malunion requiring osteotomy, nonunion, or bone loss, especially humeral shortening. Dines et al (Dines, Warren, Altchek, et al., 1993) recently reported their results with shoulder arthroplasty in twenty patients with posttraumatic changes. They emphasize the difficulty of these cases and the advisability of avoiding tuberosity osteotomy. Other series of arthroplasty for late sequellae of trauma include that of Norris et al (Norris, Green, McGuigan, 1995), Habermeyer and Schweiberer (Habermeyer and Schweiberer, 1992), and Frich et al (Frich, Sojbjerg and Sneppen, 1991).


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