Ted S. Gross, Ph.D.

Sydney T and Pea


Post-Doc University of Calgary - Joint Disease (1995)

Ph.D. SUNY Stony Brook - Mechanical Engineering (1993)

M.S. Penn State University - Sport Biomechanics (1985)

B.S. Trinity U., Texas - Engineering Science (1983)


P: 206.897.5604
F: 206.897.5611 


UW Department of Bioengineering

UW Molecular and Cellular Biology Graduate Program

American Society for Bone and Mineral Research

American Society of Biomechanics

Orthopaedic Research Society


UK Sunset


Research Interests

My most recent areas of research interest are: 1) developing strategies to make low magnitude mechanical loading osteogenic, 2) the linkage between muscle homeostasis and bone homeostasis, 3) osteocyte hypoxia as a potential signal mediator of osteoclastogenesis, and 4) the role of muscle function in fracture healing.  


Current Academic Appointments

Professor and Director
Orthopaedic Science Laboratories, Department of Orthopaedics and Sports Medicine, University of Washington (2007 to Present)

Adjunct Professor
Department of Bioengineering, University of Washington (2007 to Present)

Associate Member
Molecular and Cellular Biology Program, University of Washington (2004 to Present)



President, American Society of Biomechanics (2006)
Editorial Board, Journal of Bone and Mineral Research (2004-2010)
Secretary-Treasurer, American Society of Biomechanics (2001-2004)
Journal of Biomechanics Award, American Society of Biomechanics (2001)
Sigvard T. Hansen, Jr. Endowed Chair in Orthopaedic Traumatology (2000)
Young Scientist Award, American Society of Biomechanics (1996)
New Investigator Recognition Award, Orthopaedic Research Society (1994)
Arthritis Society/Medical Research Council Postdoctoral Fellowship (1993-95)
Alberta Heritage Medical Foundation Postdoctoral Fellowship (1993-95)
University of Calgary Postdoctoral Fellowship (1993)


Topical Publications

Poliachik, S.L., Bain, S.D., Threet, D., Huber, P. Gross, T.S. (2010).  Transient muscle paralysis disrupts bone homeostasis by rapid degradation of bone.  Bone, 46:18-23. PMID: 19857614.


Prasad J, Wiater B.P., Nork S.E., Bain S.D., Gross T.S. (2010) Characterizing gait induced normal strains in a murine tibia cortical bone defect model.  J Biomech, 43(14):2765-70. PMID: 20674920.


Srinivasan, S., Ausk B.J., Prasad J., Threet D., Bain S.D., Richardson T.S., Gross T.S. (2010).  Rescuing Loading Induced Bone Formation at Senescence.  PLoS Comput Biol, 6(9): e1000924. PMID: 20838577.


Ausk, B.J., Huber, P., Poliachik, S.L., Bain, S.D., Srinivasan, S., Gross, T.S. (2012).  Cortical bone resorption following muscle paralysis is spatially heterogeneous.  Bone, 50:14-22. PMID: 21920486.

Aliprantis, A.O., Stolina, M., Kostenuik, P.J., Poliachik, S.L., Warner, S.E., Bain, S.D., Srinivasan, S., Gross, T.S.  (2012). Transient muscle paralysis degrades bone via rapid osteoclastogenesis.  Faseb J., 26:1110-1118. PMID: 2215315.


Worton, L.E., Ausk, B.J., Downey, L.X., Bain, S.D., Gardiner, E.M., Srinivasan, S., Gross, T.S., Kwon, R.Y. (2013).  Systems-based identification of temporal processing pathways during bone cell mechanotransduction.  PLOS One, 8(9):e74205, PMID: 24040202.


Ausk, B.J., Kwon, R.Y., Bain, S.D., Srinivasan, S., Gross, T.S. (2013).  Metaphyseal and diaphyseal bone loss in the tibia following transient muscle paralysis are spatiotemporally distinct resorption events.  Bone, 57(2): 413-22, PMID: 24063948.


Srinivasan, S., Gardiner, E.M., Kwon, R.Y., Worton, L.E., Bain, S.D., Ausk, B.J., Gross, T.S. (2014).  Distinct cyclosporine A Supplements are required to enhance bone formation induced by cyclic and rest-inserted loading in the senescent skeleton.  PLOS One, 9(1):e84868, PMID: 24404194.